The Journal of Experimental Medicine
Janeway's Immunobiology 7th Edition
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Correction for Wilson and Butcher, J. Exp. Med. 200 (6) 805-809.
Published 18 October 2004. doi:10.1084/jem.20041069092904c
Rockefeller University Press, 0022-1007 $8.00
JEM, Volume 200, Number 8, 1091-1091
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Correction



Eric Wilson and Eugene C. Butcher


Vol. 200, No. 6, September 20, 2004. Pages 805–809.

Due to technical errors in the final production stages of this article, Figure 3 A was printed in black and white rather than in color. The corrected figure, along with the legend, appears below.



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Figure 3. Anti-CCL28 inhibits IgA ASC homing to the mammary gland and IgA antibody accumulation in the milk. (A) Tissue sections from the mammary gland of 9-d postpartum mice treated with anti-CCL28 function-blocking antibody or isotype control antibody. Tissue sections were stained with anti-IgA (green) and anti–TCR-ß (red) antibodies. The subiliac lymph node is included in the top region of each photograph as a reference point. A magnification of 200. (B) Milk was collected on days 1 and 9 postpartum from mice treated with anti-CCL28 or isotype control antibody. IgA, IgG1, and IgM levels in the milk were determined. Horizontal bars represent the average of each group. Differences between IgA ASCs and IgA antibody accumulation between control and anti-CCL28 treatment groups were statistically significant (P < 0.001).

 

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This Article
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